Dr Rajeev Menon. (DC)

For Dr Rajeev Menon, clinical girector, cardiology, AIG Hospitals, some cases stay with you because they force you to question what appears obvious. This was one of them.

WHEN A TRANSPLANT SEEMED THE ONLY WAY OUT

A 47-year-old woman with rheumatic heart disease had severe mitral regurgitation, leaving her heart pumping at just 20 per cent. Valve replacement was considered high-risk and a transplant was being discussed. But her unusually rapid decline raised a crucial question: Was the valve really the whole story?

THE QUESTION THAT CHANGED THE CASE

Could there be another reason? The team revisited the basics, including ECG. The simple test revealed that what initially looked like sinus tachycardia (means a faster-than-normal heartbeat, usually over 100 beats per min) was actually atrial tachycardia, an abnormal rhythm arising from the upper chambers of the heart. That finding changed everything.

WHEN A FAST HEART STARTS TO FAIL

“Atrial tachycardia can do something similar to an engine being forced to run continuously at excessive speed. An abnormal electrical focus in the upper chamber repeatedly drives the heart too fast, and when this continues for long enough, the heart muscle itself can weaken. This is what we call tachycardia-induced cardiomyopathy,” explains Dr Menon. Instead of moving directly towards transplantation, the team first decided to restore her normal rhythm.

THE MOMENT SHE REACHED FOR HIS HAND

She underwent electrical cardioversion — a controlled shock under sedation to reset the abnormal rhythm. She was understandably anxious. “At moments like these, patients rarely speak in the language of ejection fractions or arrhythmias. They speak about life. As she reached for my hand, she said, ‘Doctor, I just want to get through this and go back home to be with my children. They need me. Please do whatever you think is right.’ There is enormous trust in that sentence,” says Dr Menon. The cardioversion worked. Her normal rhythm returned and the heart began its fight back.

THEN, THE HEART BEGAN TO COME BACK

Over the following weeks, her heart began to recover. “By approximately four weeks, her ejection fraction returned to normal range. The heart that had appeared so weak that transplantation was being considered had regained its pumping ability. The mitral valve disease still needed treatment, but the situation had changed dramatically.” She’s now a much better surgical candidate.

FROM TRANSPLANT TO A TREATABLE RHYTHM

For Dr Menon, the most striking part was how one careful observation completely altered the treatment pathway. “The journey moved from the possibility of a heart transplant to correction of an abnormal rhythm, recovery of heart function and then a relatively straightforward valve replacement. And the clue was present on an ECG — a device that has been used worldwide since the 1940s,” he says.

WHY DOCTORS MUST QUESTION THE OBVIOUS

Modern cardiology has extraordinary technology, but Dr Menon believes it must never replace clinical judgement. “Technology should never replace attention. A test is only as valuable as the thought applied to it. When the clinical timeline does not make sense, we must question our first conclusion, return to the patient’s history and re-examine even the simplest investigation.”

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